Pain Aid Capsules (1500mg/10 Capsules) -Pain Killer

For chronic pain,sleep,anxiety,depression in capsules form for oral dosage

PainAID , is an Ayurvedic formulation which contains a powerful mixture of Vijaya leaf extract

and hemp seed oil. The constituents help in reduction of edema and inflammation due to their

anti-inflammatory and antioxidant effects. They also suppress any kind of pain experienced by

people due to their analgesic effects. Types of pain on which medicine works are; headaches,

period cramps, body aches due to excessive training, migraine attack, nerve pain, joint or muscle

soreness. The capsules also work by calming the cannabinoid receptors of the body to help with

insomnia, depression and anxiety.

Product Benefits:HELPS FOR

Levels Vata Dosha

Anti-inflammatory Action

Reduces Chronic pain

Brings down swelling & sensitization

Manages depression

Enhances sleep

Reduces stress and anxiety

Mechanism of action of active ingredient:

● Medical Cannabis exerts its action via two vital and active components, cannabidiol

(CBD) and delta-9-tetrahydrocannabinol (THC). THC,one of the active ingredients in

cannabis, activates cannabinoid receptors type 1 and 2 i.e CB1 and CB2 receptors (1-2).

● 2-arachidonoyl-sn-glycerol (2-AG), anandamide and endocannabinoids are produced in

injured tissues via distinct biochemical pathways that are known to suppress sensitization

and inflammation by activation of cannabinoid (CB) receptors (3-5).

● The compounds in Vijaya ultimately attach to cannabinoid receptors in the brain when

someone drinks it. These receptors have the ability to alter mental equilibrium and

produce euphoric effects. Some persons experience a decrease in depression symptoms as

a result of this change (5).

● CBD and THC, reduce pain at the site of injury.The anti-inflammatory properties of

THCs are primarily driven via activation of CB2 receptors on immune cells which

ultimately dampens the body’s pain-inducing response to injury. Whereas, CBD reduces

inflammation via blocking inflammatory mediators & shifting the activation of

macrophage repair cells from the pro-inflammatory type to the anti-inflammatory type

(6-9).

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